Kagan BL, Hirakura Y, Azimov R and Azimova R (2001) The channel hypothesis of Huntington's disease. Brain Res Bull, 56:281-4.

Hirakura Y, Azimov R, Azimova R and Kagan BL (2000) Polyglutamine-induced ion channels: a possible mechanism for the neurotoxicity of Huntington and other CAG repeat diseases. J Neurosci Res, 60:490-4.

Hirakura Y, Yiu WW, Yamamoto A and Kagan BL (2000) Amyloid peptide channels: blockade by zinc and inhibition by Congo red (amyloid channel block). Amyloid, 7:194-9.

KAGAN BL and Hirakura Y (2000) Channel formation in the pathogenesis of Alzheimer’s disease and other amyloidoses Einstein Quart. J. Biol. Med, 16:123-9.

Chen QS, Kagan BL, Hirakura Y and Xie CW (2000) Impairment of hippocampal long-term potentiation by Alzheimer amyloid beta-peptides. J Neurosci Res, 60:65-72.

Sokolov Y, Mirzabekov T, Martin DW, Lehrer RI and Kagan BL (1999) Membrane channel formation by antimicrobial protegrins. Biochim Biophys Acta, 1420:23-9.

Hirakura Y, Lin MC and Kagan BL (1999) Alzheimer amyloid abeta1-42 channels: effects of solvent, pH, and Congo Red. J Neurosci Res, 57:458-66.

Schendel SL, Azimov R, Pawlowski K, Godzik A, Kagan BL and Reed JC (1999) Ion channel activity of the BH3 only Bcl-2 family member, BID. J Biol Chem, 274:21932-6.

Mirzabekov TA, Silberstein AY and Kagan BL (1999) Use of planar lipid bilayer membranes for rapid screening of membrane active compounds. Methods Enzymol, 294:661-74.

Koo SP, Bayer AS, Kagan BL and Yeaman MR (1999) Membrane permeabilization by thrombin-induced platelet microbicidal protein 1 is modulated by transmembrane voltage polarity and magnitude. Infect Immun, 67:2475-81.

Schendel S, Azimov R, Kagan BL and Reed JC (1999) Ion channel activity of the BH3-only BCL-2 family member BID. J Biol Chem.

Kagan BL and Hirakura Y (1999) Ion channels in the pathogenesis of Alzheimer's Disease and other amyloidosis. IN: Einstein Quart. J. Biol. Med .

Kagan BL, Leskin G, Haas B, Wilkins J and Foy D (1999) Elevated lipid levels in Vietnam veterans with chronic posttraumatic stress disorder. Biol Psychiatry, 45:374-7.

Hirakura Y, Satoh Y, Hirashima N, Suzuki T, Kagan BL and Kirino Y (1998) Membrane perturbation by the neurotoxic Alzheimer amyloid fragment beta 25-35 requires aggregation and beta-sheet formation. Biochem Mol Biol Int, 46:787-94.

Shang ES, Skare JT, Exner MM, Blanco DR, Kagan BL, Miller JN and Lovett MA (1998) Isolation and characterization of the outer membrane of Borrelia hermsii. Infect Immun, 66:1082-91.

Skare JT, Mirzabekov TA, Shang ES, Blanco DR, Erdjument-Bromage H, Bunikis J, Bergstrom S, Tempst P, Kagan BL, Miller JN and Lovett MA (1997) The Oms66 (p66) protein is a Borrelia burgdorferi porin. Infect Immun, 65:3654-61.

Sellman BR, Kagan BL and Tweten RK (1997) Generation of a membrane-bound, oligomerized pre-pore complex is necessary for pore formation by Clostridium septicum alpha toxin. Mol Microbiol, 23:551-8.

Lin MC, Mirzabekov T and Kagan BL (1997) Channel formation by a neurotoxic prion protein fragment. J Biol Chem, 272:44-7.

Skare JT, Champion CI, Mirzabekov TA, Shang ES, Blanco DR, Erdjument-Bromage H, Tempst P, Kagan BL, Miller JN and Lovett MA (1996) Porin activity of the native and recombinant outer membrane protein Oms28 of Borrelia burgdorferi. J Bacteriol, 178:4909-18.

Mirzabekov TA, Lin MC and Kagan BL (1996) Pore formation by the cytotoxic islet amyloid peptide amylin. J Biol Chem, 271:1988-92.

Kagan BL, Ganz T and Lehrer RI (1994) Defensins: a family of antimicrobial and cytotoxic peptides. Toxicology, 87:131-49.

Kagan BL, Baldwin RL, Munoz D and Wisnieski BJ (1992) Formation of ion-permeable channels by tumor necrosis factor-alpha. Science, 255:1427-30.

Hirakura Y and KAGAN BL ((in press) 2000) Pore formation by Beta-2-Microglobulin: a mechanism for the pathogenesis of dialysis associated amyloidosis Amyloid: Int J Clin Experience Research.

Mirzabekov TA, Silberstein AV, Hirakura Y and KAGAN BL ((in press) 1999) A new chamber for rapid screening of membrane active compounds Biophysics (JAPAN).

Hirakura Y and KAGAN BL (in review 2000) Serum Amyloid A forms ion permeable channels in planar lipid bilayer membranes .

Schein, S.J., Kagan, B.L., Finkelstein, A. (1978). Colicin K acts by forming voltage dependent channels in phospholipid bilayer membranes. Nature, 276:159-163.

Weaver, C.A., Kagan, B.L., Finkelstein, A., Konisky, J. (1981). Mode of action of colicin Ib: formation of ion-permeable membrane channels. Biochim. Biophys. Acta, 645:137-142.

Kagan, B.L., Finkelstein, A., Colombini, M. (1981). Diphtheria toxin fragments forms large pores in phospholipid bilayer membranes. Proc. Nat. Acad. Sci. USA, 78:4950-4954.

Kagan, B.L. (1982). Voltage dependent channels formed by colicins. Ph.D. Thesis. Yeshiva University, New York, NY.

Kagan, B.L., Pollard, H.B., Hanna, R.B. (1982).Ion-permeable channels induced by the venom of the fanged bloodworm Glycera dibranchiata. Toxicon., 20:887-893.

Kagan, B.L. (1983). Mode of action of yeast killer toxins: Channel formation in lipid bilayer membranes. Nature, 302:709-711.

Kagan, B.L., Reich, K.A., Collier, R.J. (1984). Orientation of the diphtheria toxin channel in lipid bilayers. Biophys. J., 45:102-104.

Roa, M., Kagan, B.L., Boquet, P. (1986). Structure/function relationships of tetanus toxin. A comparison with the diphtheria toxin molecule. In: Bacterial Protein Toxins. P. Falmagne, J.E. Alouf, F.J. Fehrenbach, J. Jeljaszewics and M. Thelestem, eds. Second European Workshop, Gustav Fischer, Stuttgart-New York, pp. 27-32.

Altshuler, L.L., Kagan, B.L., Baxter, Jr., L.R., Smith, G., Wilkins, J.N. (1987). Effect of interrupted sleep patterns and partial sleep deprivation on DST and mood in psychiatric house officers. Acta Psych. Scand., 75:614-618.

Kagan, B.L. (1987). Lysosomotropic agents in the treatment of AIDS. Western J. Med., 146:234.

Kagan, B.L. (1989). A molecular mechanism of panic attacks. Spec. Sci. Tech., 12:199-208.

Weinstein, H. C., Hoover, J. O., Kagan, B.L., Metzner, J.L., Sadoff, R.L., Zimmerman, V.H. (1989). Psychiatric services in jails and prisons. Task Force Report, 29:1-44.

Kagan, B.L., Selsted, M.D., Ganz, T., Lehrer, R.I. (1990). Antimicrobial defensin peptides form voltage-dependent ion-permeable channels in planar lipid bilayer membranes. Proc. Nat. Acad. Sci. USA, 87:210-214.

Kagan, B.L., Sultzer, D., Rosenlicht, N., Gerner, R.H. (1990).Oral S-adenosyl methionine in depression: a double-blind placebo controlled trial. Am. J.Psych.,147:591-595.

Kagan, B.L. (1990) Methylation, folate metabolism, and S-adenosyl-methionine in psychiatric disorders. Psychiatry:A world perspective, 3:424-427.

Kagan, B.L., Sultzer, D., Rosenlicht, N., Gerner, R.H. (1990) Oral S-adenosyl-methionine in the treatment of depression. Psychiatry: A world perspective, 3:428-433.

Krantz, D.E., Zidovetzki, R., Kagan, B.L., Zipursky, S.L. (1991). Amphipathic beta structure of a leucine-rich repeat. J. Biol. Chem., 206:16801-16807.

Kagan, B.L. (1991) Inositol 1,4,5 triphosphate directly opens diphtheria toxin channels. Biochim. Biophys. Acta, 1069:145-150.

Kagan, B.L., Baldwin, R.L., Munoz, D. Wisniewski, B.. (1992). Tumor necrosis factor-alpha forms ion-permeable channels. Science, 255:1427-1430.

Marmar, C., Foy, D., Kagan, B.L., Pynoos, R. (1993). An integrated treatment approach to post traumatic stress disorder. Ann. Rev. Psych., 13: 12:243-268.

Kagan, B.L., Mirzabekov, T., Munoz, D., Baldwin, R.L., Wisnieski, B., (1993) The Role of Channel Formation in the Mechanism of Action of Tumor Necrosis Factors. Ann NY Acad Sci, 707:317-327.

Tweten, R., Ballard, J., Sokolov, Y., Yuan, W.L., Kagan, B.L. (1993) Activation and Mechanism of Clostridium septicum alpha toxin. Molecular Microbiology, 10:627-634.

Kagan, B.L., Sokolov, Y. (1994). Planar lipid bilayer membranes as an assay for pore forming toxins. In: Methods in Enzymology - Bacterial Pathogenesis (Part A). V.L. Clark and P.M. Bavoil, eds. Vol. 235:699-713.

Kagan, B.L., Ganz, T., Lehrer, R. (1994). Defensins: A family of antimicrobial and cytotoxic peptides. Toxicology, 87:131-149.

Mirzabekov, T., Lin, M.,Yuan, W.L., Marshall, P. Carman, M., Tomaselli, K., Lieberberg, I., Kagan, B.L. (1994). Channel Formation by a Neurotoxic Fragment of the Beta-Amyloid Peptide, Bioch. Biophys. Res. Com. 202:1142-1148.

Baldwin, R.L., Mirzabekov, T., Kagan, B.L., Wisnieski, B., (1995). The conformation and ion channel activity of Lymphotoxin at neutral and low pH. J. Immunol. 154:790-798.

Skare, J.T., Shang, E.S., Foley, D.M., Blanco, D.R., Champion, C.I., Mirzabekov, T., Sokolov, Y., Kagan, B.L., Miller, J.N., Lovett, M.A. (1995) Virulent Strain-Associated Outer Membrane Proteins of Borrelia burgdorferi. J. Clin. Invest. 96:2380-2392.

Mirzabekov, T., Lin, M., Kagan, B.L. (1996). Pore Formation by the Cytotoxic Islet Amyloid Peptide Amylin. J. Biol.Chem.271:1988-1992.

Lin, M., Mirzabekov, T., Kagan, B.L. (1996) Channel Formation by a Neurotoxic Beta Amyloid Peptide, AB 25-35 in Neurodegenerative Disease, Fiskum, G., ed. Plenum Press, New York.

Skare, J.T., Champion, C. I., Mirzabekov, T.A., Shang, E.S., Blanco, D.R., Erdjument-Bromage, M., Tempst, P., Kagan, B.L., Miller, J.N., Lovett, M.A. (1996). Porin activity of the native and recombinant outer membrane protein Oms 28 of Borrelia burgdorferi. J. Bacteriol. 178:4909-18.

Lin, M., Mirzabekov, T., Kagan, B.L. (1997). Channel formation by a neurotoxic fragment of the prion protein. J.Biol Chem. 272:44-47.

Lin, M., Kagan, B.L. (1997) Giant multilevel cation channels formed by Alzheimer's Disease Amyloid b-protein ABP(1-40)] in bilayer membranes. Chemtracts 10:251-254.

Sellman, B.R., Kagan, B.L., Tweten, R.K. (1997) Generation of a membrane-bound, oligomerized pre-pore complex is necessary for pore formation by Clostridium septicum alpha toxin. Molecular Microbiology 23, 551-8.

Skare, J.T., Mirzabekov, T. A., Shang, E.S., Blanco, D.R., Bunikis, J., Erdjument-Bromage, H., Tempst, P., Weinberg, A., Bergstrom, S., Kagan, B.L., Miller, J.N., and Lovett, M. A. (1997) The Oms66 (p66) protein function is a porin. Inf. Imm 65, 3654-61.

Shang, E.S., Skare, J.T., Exner, M.M., Blanco, D.R., Kagan, B.L., Miller, J.N., Lovett, M.A.(1998). Isolation and characterization of the outer membrane of Borrelia hermsii. Inf. Imm. 66,1082-91.

Foy, D., Kagan, B.L., McDermott, C., Leskin, G., Sipprelle, R.C., Paz, G. (1998) Practical Parameters in the use of Flooding for Treating Chronic PTSD. Clinical Psychology and Psychotherapy: An International Journal of Theory and Practice.

Mirzabekov, T., Silberstein, A., Kagan, B.L. (1998) The use of planar lipid bilayer membranes for rapid screening of membrane active compounds. Meth Enz. 294:61-74.

Kagan, B.L., Leskin, G.,Haas, B., Wilkins, J., Foy, D. (1999). Elevated lipid levels in chronic Post-Traumatic Stress Disorder. Biol. Psych. 45:374-7.

Hirakura, Y. Satoh, Y., Hirashima, N., Suzuki, T., Kagan, B. L., Kirino, Y. (1999). Membrane perturbation by the neurotoxic Alzheimer amyloid fragment b 25-35 requires aggregation and b-sheet formation. Biochemistry and Molecular Biology International. 46:787-94.

Koo, S.P., Bayer, A.S., KAGAN, B.L., Yeaman, M.R. (1999). Membrane permeabilization by Thrombin-induced Platelet Microbicidal Protein-1 is Modulated by Transmembrane Voltage Polarity and Magnitude. Inf. Imm. 67:2475-81.

Hirakura, Y., Lin, M-C., Kagan, B. L. (1999). Alzheimer amyloid Ab 1-42 channels: Effects of solvent, pH, and Congo red. J. Neurosci. Res. 57:458-66.

Sokolov, Y., Mirzabekov, T.A., Martin, D., Lehrer, R. L., Kagan, B.L. (1999). Membrane disruption and channel formation by antimicrobial protegrins. Bioch. Biophys. Acta, 1420:23-9.

Schendel, S.L., Azimov, R., Panlowski, K., Godzik, A., KAGAN, B.L., Reed, J.C. (1999). Ion channel activity of the BH3 only BCL-2 family member, BID. J. Biol. Chem. 274:21932-6.

Mirzabekov, T.A., Silberstein, A.V., Hirakura, Y., KAGAN, B.L. (1999). A new chamber for rapid screening of membrane active compounds. Biophysics (JAPAN), (in press).

Kagan, B.L., Hirakura, Y. (2000). Channel formation in the pathogenesis of Alzheimer’s disease and other amyloidoses. Einstein Quart. J. Biol. Med. 16:123-9.

Chen, Q-S., KAGAN, B.L., Hirakura, Y., Xie, C.-W. (2000). Impairment of hippocampal long-term potentiation by Alzheimer Amyloid- Ab Peptides. J. Neurosci. Res. 60:65-72.

Hirakura, Y., Azimov, R., Azimova, R., KAGAN, B.L. (2000) Polyglutamine induced ion channels: a possible mechanism for the neurotoxicity of Huntington’s and other CAG repeat diseases. J. Neurosci Res 60:490-4.

Hirakura, Y, Yiu, W., Yamamoto, A., KAGAN, B. L. (2000) Amyloid peptide channels: Blockade by zinc and inhibition by Congo Red. Amyloid: Int J Exp Clin Res 7: 194-199.

Hirakura, Y., KAGAN, B.L. (2001). Pore formation by Beta-2-Microglobulin: a mechanism for the pathogenesis of dialysis associated amyloidosis. Amyloid: J Prot Fold Dis 8, 94-100.

Hirakura, Y., Azimova, R., Azimov, R., KAGAN, B.L. (2001). The channel hypothesis of Huntington’s disease. Brain Res Bull 56, 281-284.

Hirakura, Y., KAGAN, B.L. (2001). Channel formation in the pathogenesis of Alzheimer's disease and other amyloidosis. Food and Food Ingredients Journal of Japan. 192:20-26.

Hirakura, Y., KAGAN, B.L. (2002). The Channel Hypothesis of Amyloidosis. Cellular and Molecular Mechanics of Toxin Action, Vol. 5. (in press).

Hirakura, Y., Carreras, I., Sipe, J.L., Kagan, B. L. (2002). Channel Formation by Serum Amyloid: a potential mechanism for amyloid pathogenesis and host defense. Amyloid 9,13-23.

Lin, M.C., Mirzabekov, T., Kagan, B.L. (2002) Electrophysiologic properties of Ab 25-35 channels. Peptides (in press).

Hirakura, Y., Lin, M-C., KAGAN, B.L. (2002). The channel hypothesis of Alzheimer's disease. Peptides (in press).

Shang, E.S., Skare J.T., Mirzabekov, T., Blanco, D.R., Kagan, B.L., Miller, J.N., Lovett, M.A. (2002). Isolation and purification of a 45 kDa outer membrane porin protein from Borrelia burgdorferi. (in review).

Rosenberg, Y., Zhang, Y.L., Aguilar-Carreno, H., Mirzabekov, T., Januszeski, M., Hubbell, W., Kagan, B.L., Anderson, W.F. (2002) Viral entry: Membrane destabilization by cytoplasmic amphiphilic and helix of envelope protein. (in review)

Lin, M.C., Mirzabekov, T., Glabe, C., Cotman, C., Kagan, B.L., (2002). Channel activity of Ab 25-35 variants is necessary but not sufficient for neurotoxicity. (in review).

Yuan, W.L., Kagan, B.L. (2002). Chloride gradients inhibit diphtheria toxin channel gating. (in preparation).

Kagan, B.L., Foy, D.W., Leskin, G. (2002). Altered Thyroid Function in Post-traumatic stress disorder. (in preparation).

Kagan, B.L., Rosenlicht, N., Herbst, M., Gerner, R.H. (2002) Folate levels in depression. (in preparation).

Kagan, B.L., Taghavi, B. (2002). Channels formed by Pseudomonas exotoxin A. (in preparation).

Leskin, G., Foy, D. W., Kagan, B.L., (2002). Long term follow up of flooding in combat related PTSD. (in preparation).

Yuan, W.L., Kagan, B.L. (2002). Cadmium inhibition of diphtheria toxin channel gating and translocation. (in preparation).

Kagan, B.L., Finkelstein, A. (1979). Channels formed in planar bilayer membranes by mutant diphtheria toxin molecule. Biophys. J., 25:181a.

Kagan, B.L., Varanda, W., Finkelstein, A. (1980). Colicin channels as a model for studying electrical excitability.Fed. Proc., 39:ix.

Kagan, B.L., Pollard, H.B., Hanna, R.B. (1980). An ion-permeable channel produced by venom of the f anged bloodworm Glycera dibranchita. Biol. Bull., 159:485.

Kagan, B.L., Finkelstein, A. (1982). Channels formed by the killer toxin of the yeast Pichia kluyveri. Biophys. J., 37:208a.

Misler, S., Kagan, B.L., Finkelstein, A. (1982). Single channel conductances formed by diphtheria toxin fragments in planar bilayers. Biophys. J., 37:323a.

Kagan, B.L. (1987). Inositol 1,4,5 triphosphate opens diphtheria toxin channels. Biophys. J. 51:250a.

Kagan, B.L., Selsted, M.E., Lehrer, R.I. (1987). A neutrophil cytotoxic peptide (defensin) increases the anion permeability of lipid bilayers. Clin. Res., 30:60a.

Kagan, B.L. (1988). Pseudomonas exotoxin A forms pH-dependent channels. Biophys. J., 53:513a.

Selsted, M.E., Pardi, A., Kagan, B.L. (1988). The three dimensional structure of a human neutrophil defensin:implications for membrane channel formation and microbicidal mechanism. Blood, 72(1):152a.

Rosenlicht, N.Z., Kagan, B.L., Sultzer, D.L., Gerner, R.H. (1989). Oral S-adenosyl-methionine as an antidepressant. J. Clin. Pharmacol., 29:839.

Yuan, W.L., Kagan, B.L. (1991). Cadmium selectively inhibits gating and translocation in the diphtheria toxin channel. Biophys. J., 59:459a.

Yuan, W.L., Kagan, B.L., (1992). Chloride gradients inhibit channel formation by diphtheria toxin. Biophys. J. 61:2975.

Sokolov, Y., Kagan, B.L., Fimmel, C., Wickner, W. The reconstitution of E. coli protein translocase in lipid bilayer membranes. Biophys. J. (1992), 61:477.

Kagan, B.L., Haas, B. (1992) High Cholesterol Levels in Chronic Post Traumatic Stress Disorder. Presented at Annual Meeting of International Society for Traumatic Stress Studies (San Antonio, Texas).

Gies, M., Haas, B., Kagan, B.L. (1992) Fluoxetine in the treatment of chronic Post Traumatic Stress Disorder. J. Traumatic Stress. Presented at Annual Meeting of International Society for Traumatic Stress Studies (San Antonio, Texas).

Baldwin, R.L., Kagan, B.L., Munoz, D., Wisnieski, B.J., (1992). TNF forms ion permeable channels. European Cytokine Netw. 3: 180.

Marshall, P., Mirzabekov, T., Yuan, W.L., Carman, M., Lieberberg, T., Kagan B.L., (1993). Channels in lipid bilayers induced by a fragment of the ?-Amyloid peptide. Biophys. J.64:A94.

Sokolov, Y., Kagan B.L., Yuan, W.L., Tweeten, R.K., Ballard, J. (1993). Channels produced by Clostridium Septicum ?-toxin in lipid bilayers. Biophys. J.64:A185.

Kagan, B.L., Foy, D., Haas, B., Hammen, M., Reno, S., Moore, R., McDermott, C. (1993) Long term effects of flooding therapy in chronic Post-traumatic stress disorder patients. Present at Annual Meeting of International Society for Traumatic Stress Studies (Los Angeles, CA).

Mirzabekov, T., Lin, M., Kagan, B.L.(1994). Ion permeability properties of lipid bilayers containing amyloid beta peptide and its fragments. Biophys. J.,66:A430

Wisniewski, B.J., Baldwin, R.L., Sinha, S., Mirzabekov, T., Kagan, B.L. (1995) The Structures and Channel Activities of Tumor Necrosis Factor alpha and beta at Neutral and Low pH. Biophys. J. 68:A203.

Munoz, D.M., Sokolov, Y., Ganz, T., Lehrer, R.I., Kagan, B.L. 1995). Channel forming activity of human defensins dependence on N-terminal residue. Biophys J. 68,A222

Lehrer, R.I. Panyutich, E., Oren, A., Sokolov, Y., Cho, Y., Harwig, S.S.L., Kagan, B.L. (1995). Protegrins: mechanisms of bactericidal activity against Gram-negative bacteria. J. Invest. Med. 43(Suppl.2):288A.

Lin, M., Mirzabekov, T., Kagan, B.L. (1995) Channel Forming Activity of Amyloidogenic Peptides. Biophys J. 68:A383.

Mirzabekov, T., Lin, M., Kagan, B.L.(1996) Amylin Induces Ion Channels in Phospholipid Membranes. Biophys. J. 70:A195

Lin, M., Mirzabekov, T., Kagan, B.L.(1996) Pore Formation by Neurotoxic Fragment of the Prion Protein.Biophys J. 70:A408

Mirzabekov, T., Skare, J.T., Shang, E. S., Blanco, D.R., Miller, J.N., Lovett, M.A., Kagan, B.L. (1996) Electrical properties of porins from Borrelia burgdorferi 70:A205.

Lin, M., Cotman, C., Rydell, R.,Kagan, B.L. (1997) Channel formationcorrelates with neurotoxicity of amyloid peptides. Soc. Neurosci. Abst. (in press).

Hirakura, Y., Kagan, B.L. (1998) Channel formation by Ab1-42 in planar lipid bilayers. Biophys J. (in press).

Chen, O., Kagan, B.L., Xie, C-W. (1998) Inhibition of hippocampal LTP by amyloid-b-peptide. Neurosci Abst. (in press).

Koo, S.P., Kagan, B.L., Yeaman, M. R., Bayer, A.S. (1998). Membrane Permeabilization by Thrombin-Induced Platelet Microbicidal Protein-1 (tPMP-1) is Modulated by Transmembrane Voltage Orientation and Magnitude. Antimicrob Ag and Chemo (in press).

Azimov, R., Azimova, R, Hirakura, Y., KAGAN, B.L. (2001). Channel formation by transthyretin. Biophys J. (in press).